TB-500 Fragment 17-23

TB-500 Fragment 17-23 (sequence: LKKTETQ) represents the minimal actin-binding domain of Thymosin Beta-4. This 7-amino-acid fragment retains the ability to sequester G-actin and promote cell migration and wound healing.

Category: Healing / Research. Evidence rating: D (animal/preclinical only).

Clinical status: Research-only

Sequesters G-actin monomers, promoting actin polymerization and cytoskeletal reorganization. This drives cell migration, angiogenesis, and wound healing at the cellular level.

Safety considerations: No specific safety data for this fragment.

Reviewed by the PeptideAtlas Editorial Team.

TB-500 Fragment 17-23 — measured properties

Production methodsynthetic
Anti-doping statusProhibited in sport (WADA)

Frequently asked questions

Is this fragment better than full TB-500?

The fragment contains the key actin-binding domain, but the full peptide may have additional activities from other domains. No head-to-head studies exist.

What is TB-500 Fragment 17-23?

TB-500 Fragment 17-23 (sequence: LKKTETQ) represents the minimal actin-binding domain of Thymosin Beta-4. This 7-amino-acid fragment retains the ability to sequester G-actin and promote cell migration and wound healing.

How strong is the evidence for TB-500 Fragment 17-23?

On the PeptideAtlas A–F scale, TB-500 Fragment 17-23 is rated D (Preclinical Only). Mostly animal or preclinical evidence

What is the clinical status of TB-500 Fragment 17-23?

Research-only

How does TB-500 Fragment 17-23 work?

Sequesters G-actin monomers, promoting actin polymerization and cytoskeletal reorganization. This drives cell migration, angiogenesis, and wound healing at the cellular level.

Coverage on this site: How Is TB-500 Handled in Research Settings?.